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Loss of KCNJ10 protein expression abolishes endocochlear potential and causes deafness in Pendred syndrome mouse model

  • Philine Wangemann1Email author,
  • Erin M Itza1,
  • Beatrice Albrecht1,
  • Tao Wu1,
  • Sairam V Jabba1,
  • Rajanikanth J Maganti1,
  • Jun Ho Lee1,
  • Lorraine A Everett2,
  • Susan M Wall3,
  • Ines E Royaux2,
  • Eric D Green2 and
  • Daniel C Marcus1
BMC Medicine20042:30

DOI: 10.1186/1741-7015-2-30

Received: 16 April 2004

Accepted: 20 August 2004

Published: 20 August 2004

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Open Peer Review reports

Pre-publication versions of this article and author comments to reviewers are available by contacting

Original Submission
16 Apr 2004 Submitted Original manuscript
Resubmission - Version 2
Submitted Manuscript version 2
Reviewed Reviewer Report
Reviewed Reviewer Report
Resubmission - Version 3
Submitted Manuscript version 3
20 Aug 2004 Editorially accepted
20 Aug 2004 Article published 10.1186/1741-7015-2-30

How does Open Peer Review work?

Open peer review is a system where authors know who the reviewers are, and the reviewers know who the authors are. If the manuscript is accepted, the named reviewer reports are published alongside the article. Pre-publication versions of the article and author comments to reviewers are available by contacting All previous versions of the manuscript and all author responses to the reviewers are also available.

You can find further information about the peer review system here.

Authors’ Affiliations

Anatomy & Physiology Department, Kansas State University
Genome Technology Branch, National Human Genome Research Institute, National Institutes of Health
Department Medicine, Renal Division, Emory University, School of Medicine